Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 220 entries
Sorted by: Best Match Show Resources per page
Cytoprotective potential of tiron and methyl palmitate against acetaminophen-induced acute liver injury.

Canadian journal of physiology and pharmacology

Shoeib AM, Said E, Ammar EM.
PMID: 26569614
Can J Physiol Pharmacol. 2016 Feb;94(2):147-154. doi: 10.1139/cjpp-2015-0270. Epub 2015 Jul 20.

Acute liver injury is a debilitating disorder associated with loss of synthetic and detoxifying functions of the liver. This investigation was designed to assess cytoprotective efficacy of daily oral tiron (300 mg/kg) and daily oral methyl palmitate (300 mg/kg)...

Cite
Shoeib AM, Said E, Ammar EM. Cytoprotective potential of tiron and methyl palmitate against acetaminophen-induced acute liver injury. Can J Physiol Pharmacol. 2015;94(2):147-154doi: 10.1139/cjpp-2015-0270.
Shoeib, A. M., Said, E., & Ammar, E. M. (2016). Cytoprotective potential of tiron and methyl palmitate against acetaminophen-induced acute liver injury. Canadian journal of physiology and pharmacology, 94(2), 147-154. https://doi.org/10.1139/cjpp-2015-0270
Shoeib, Amal M, et al. "Cytoprotective potential of tiron and methyl palmitate against acetaminophen-induced acute liver injury." Canadian journal of physiology and pharmacology vol. 94,2 (2016): 147-154. doi: https://doi.org/10.1139/cjpp-2015-0270
Shoeib AM, Said E, Ammar EM. Cytoprotective potential of tiron and methyl palmitate against acetaminophen-induced acute liver injury. Can J Physiol Pharmacol. 2016 Feb;94(2):147-154. doi: 10.1139/cjpp-2015-0270. Epub 2015 Jul 20. PMID: 26569614.
Copy Download .nbib Format: NLM
Relationship between male hormonal status, Bezold-Jarisch reflex function, and ACE activity (cardiac and plasmatic).

Canadian journal of physiology and pharmacology

Heringer OA, Cassaro KODS, Barbosa NCMR, Brasil GA, do Nascimento AM, de Lima EM, Bissoli NS, Lenz D, Endringer DC, de Andrade TU.
PMID: 26641722
Can J Physiol Pharmacol. 2016 Feb;94(2):231-236. doi: 10.1139/cjpp-2015-0144. Epub 2015 Aug 24.

The negative relationship between androgens and the Bezold-Jarisch reflex (BJR) has been demonstrated, but no studies evaluated the physiological influence of testosterone on this reflex. We evaluated the influence of male rat castration on the BJR, cardiac morphometric parameters,...

Cite
Heringer OA, Cassaro KODS, Barbosa NCMR, et al. Relationship between male hormonal status, Bezold-Jarisch reflex function, and ACE activity (cardiac and plasmatic). Can J Physiol Pharmacol. 2015;94(2):231-236doi: 10.1139/cjpp-2015-0144.
Heringer, O. A., Cassaro, K. O. D. S., Barbosa, N. C. M. R., Brasil, G. A., do Nascimento, A. M., de Lima, E. M., Bissoli, N. S., Lenz, D., Endringer, D. C., & de Andrade, T. U. (2016). Relationship between male hormonal status, Bezold-Jarisch reflex function, and ACE activity (cardiac and plasmatic). Canadian journal of physiology and pharmacology, 94(2), 231-236. https://doi.org/10.1139/cjpp-2015-0144
Heringer, Otávio Arruda, et al. "Relationship between male hormonal status, Bezold-Jarisch reflex function, and ACE activity (cardiac and plasmatic)." Canadian journal of physiology and pharmacology vol. 94,2 (2016): 231-236. doi: https://doi.org/10.1139/cjpp-2015-0144
Heringer OA, Cassaro KODS, Barbosa NCMR, Brasil GA, do Nascimento AM, de Lima EM, Bissoli NS, Lenz D, Endringer DC, de Andrade TU. Relationship between male hormonal status, Bezold-Jarisch reflex function, and ACE activity (cardiac and plasmatic). Can J Physiol Pharmacol. 2016 Feb;94(2):231-236. doi: 10.1139/cjpp-2015-0144. Epub 2015 Aug 24. PMID: 26641722.
Copy Download .nbib Format: NLM
Pretreatment with Astragaloside IV protects human umbilical vein endothelial cells from hydrogen peroxide induced oxidative stress and cell dysfunction via inhibiting eNOS uncoupling and NADPH oxidase - ROS - NF-κB pathway.

Canadian journal of physiology and pharmacology

Xu C, Tang F, Lu M, Yang J, Han R, Mei M, Hu J, Wang H.
PMID: 27453997
Can J Physiol Pharmacol. 2016 Nov;94(11):1132-1140. doi: 10.1139/cjpp-2015-0572. Epub 2016 May 30.

Endothelial cell injury caused by reactive oxygen species (ROS) plays a critical role in the pathogenesis of cardiovascular disorders. Astragaloside IV (AsIV) possesses potent antioxidant properties against oxidative stress through undefined mechanism(s). We sought to investigate whether AsIV protects...

Cite
Xu C, Tang F, Lu M, et al. Pretreatment with Astragaloside IV protects human umbilical vein endothelial cells from hydrogen peroxide induced oxidative stress and cell dysfunction via inhibiting eNOS uncoupling and NADPH oxidase - ROS - NF-κB pathway. Can J Physiol Pharmacol. 2016;94(11):1132-1140doi: 10.1139/cjpp-2015-0572.
Xu, C., Tang, F., Lu, M., Yang, J., Han, R., Mei, M., Hu, J., & Wang, H. (2016). Pretreatment with Astragaloside IV protects human umbilical vein endothelial cells from hydrogen peroxide induced oxidative stress and cell dysfunction via inhibiting eNOS uncoupling and NADPH oxidase - ROS - NF-κB pathway. Canadian journal of physiology and pharmacology, 94(11), 1132-1140. https://doi.org/10.1139/cjpp-2015-0572
Xu, Chonghua, et al. "Pretreatment with Astragaloside IV protects human umbilical vein endothelial cells from hydrogen peroxide induced oxidative stress and cell dysfunction via inhibiting eNOS uncoupling and NADPH oxidase - ROS - NF-κB pathway." Canadian journal of physiology and pharmacology vol. 94,11 (2016): 1132-1140. doi: https://doi.org/10.1139/cjpp-2015-0572
Xu C, Tang F, Lu M, Yang J, Han R, Mei M, Hu J, Wang H. Pretreatment with Astragaloside IV protects human umbilical vein endothelial cells from hydrogen peroxide induced oxidative stress and cell dysfunction via inhibiting eNOS uncoupling and NADPH oxidase - ROS - NF-κB pathway. Can J Physiol Pharmacol. 2016 Nov;94(11):1132-1140. doi: 10.1139/cjpp-2015-0572. Epub 2016 May 30. PMID: 27453997.
Copy Download .nbib Format: NLM
The functional morphology and role of cardiac telocytes in myocardium regeneration.

Canadian journal of physiology and pharmacology

Varga I, Danisovic L, Kyselovic J, Gazova A, Musil P, Miko M, Polak S.
PMID: 27428566
Can J Physiol Pharmacol. 2016 Nov;94(11):1117-1121. doi: 10.1139/cjpp-2016-0052. Epub 2016 May 19.

Key morphological discoveries in recent years have included the discovery of new cell populations inside the heart called cardiac telocytes. These newly described cells of the connective tissue have extremely long cytoplasmic processes through which they form functionally connected...

Cite
Varga I, Danisovic L, Kyselovic J, et al. The functional morphology and role of cardiac telocytes in myocardium regeneration. Can J Physiol Pharmacol. 2016;94(11):1117-1121doi: 10.1139/cjpp-2016-0052.
Varga, I., Danisovic, L., Kyselovic, J., Gazova, A., Musil, P., Miko, M., & Polak, S. (2016). The functional morphology and role of cardiac telocytes in myocardium regeneration. Canadian journal of physiology and pharmacology, 94(11), 1117-1121. https://doi.org/10.1139/cjpp-2016-0052
Varga, Ivan, et al. "The functional morphology and role of cardiac telocytes in myocardium regeneration." Canadian journal of physiology and pharmacology vol. 94,11 (2016): 1117-1121. doi: https://doi.org/10.1139/cjpp-2016-0052
Varga I, Danisovic L, Kyselovic J, Gazova A, Musil P, Miko M, Polak S. The functional morphology and role of cardiac telocytes in myocardium regeneration. Can J Physiol Pharmacol. 2016 Nov;94(11):1117-1121. doi: 10.1139/cjpp-2016-0052. Epub 2016 May 19. PMID: 27428566.
Copy Download .nbib Format: NLM
Too much impact for the impact factor: are a new generation of scientists in peril?.

Canadian journal of physiology and pharmacology

[No authors listed]
PMID: 22423891
Can J Physiol Pharmacol. 2012 Apr;90(4):iii-iv. doi: 10.1139/y2012-021. Epub 2012 Mar 16.

No abstract available.

Cite
Too much impact for the impact factor: are a new generation of scientists in peril?. Can J Physiol Pharmacol. 2012;90(4):iii-ivdoi: 10.1139/y2012-021.
(2012). Too much impact for the impact factor: are a new generation of scientists in peril?. Canadian journal of physiology and pharmacology, 90(4), iii-iv. https://doi.org/10.1139/y2012-021
"Too much impact for the impact factor: are a new generation of scientists in peril?." Canadian journal of physiology and pharmacology vol. 90,4 (2012): iii-iv. doi: https://doi.org/10.1139/y2012-021
Too much impact for the impact factor: are a new generation of scientists in peril?. Can J Physiol Pharmacol. 2012 Apr;90(4):iii-iv. doi: 10.1139/y2012-021. Epub 2012 Mar 16. French. PMID: 22423891.
Copy Download .nbib Format: NLM
The Most Highly Cited Papers in the Canadian Journal of Physiology and Pharmacology Through the Decades.

Canadian journal of physiology and pharmacology

[No authors listed]
PMID: 22762125
Can J Physiol Pharmacol. 2012 Jul;90(7):iii-iv. doi: 10.1139/y2012-113. Epub 2012 Jul 04.

No abstract available.

Cite
The Most Highly Cited Papers in the Canadian Journal of Physiology and Pharmacology Through the Decades. Can J Physiol Pharmacol. 2012;90(7):iii-ivdoi: 10.1139/y2012-113.
(2012). The Most Highly Cited Papers in the Canadian Journal of Physiology and Pharmacology Through the Decades. Canadian journal of physiology and pharmacology, 90(7), iii-iv. https://doi.org/10.1139/y2012-113
"The Most Highly Cited Papers in the Canadian Journal of Physiology and Pharmacology Through the Decades." Canadian journal of physiology and pharmacology vol. 90,7 (2012): iii-iv. doi: https://doi.org/10.1139/y2012-113
The Most Highly Cited Papers in the Canadian Journal of Physiology and Pharmacology Through the Decades. Can J Physiol Pharmacol. 2012 Jul;90(7):iii-iv. doi: 10.1139/y2012-113. Epub 2012 Jul 04. PMID: 22762125.
Copy Download .nbib Format: NLM
Retraction: Mitigation of azathioprine-induced testicular atrophy by taurine; an impact on inflammation, oxidative perturbations and apoptosis.

Canadian journal of physiology and pharmacology

Mohammad SA, Ramadan BK, Schaalan MF.
PMID: 29493317
Can J Physiol Pharmacol. 2018 Mar;96(3):318. doi: 10.1139/cjpp-2018-0072.

No abstract available.

Cite
Mohammad SA, Ramadan BK, Schaalan MF. Retraction: Mitigation of azathioprine-induced testicular atrophy by taurine; an impact on inflammation, oxidative perturbations and apoptosis. Can J Physiol Pharmacol. 2018;96(3):318doi: 10.1139/cjpp-2018-0072.
Mohammad, S. A., Ramadan, B. K., & Schaalan, M. F. (2018). Retraction: Mitigation of azathioprine-induced testicular atrophy by taurine; an impact on inflammation, oxidative perturbations and apoptosis. Canadian journal of physiology and pharmacology, 96(3), 318. https://doi.org/10.1139/cjpp-2018-0072
Mohammad, Salwa A, et al. "Retraction: Mitigation of azathioprine-induced testicular atrophy by taurine; an impact on inflammation, oxidative perturbations and apoptosis." Canadian journal of physiology and pharmacology vol. 96,3 (2018): 318. doi: https://doi.org/10.1139/cjpp-2018-0072
Mohammad SA, Ramadan BK, Schaalan MF. Retraction: Mitigation of azathioprine-induced testicular atrophy by taurine; an impact on inflammation, oxidative perturbations and apoptosis. Can J Physiol Pharmacol. 2018 Mar;96(3):318. doi: 10.1139/cjpp-2018-0072. PMID: 29493317.
Copy Download .nbib Format: NLM
Different blood pressure responses to opioids in 3 rat hypertension models: role of the baseline status of sympathetic and renin-angiotensin systems.

Canadian journal of physiology and pharmacology

Bądzyńska B, Lipkowski AW, Olszyński KH, Sadowski J.
PMID: 27494747
Can J Physiol Pharmacol. 2016 Nov;94(11):1159-1169. doi: 10.1139/cjpp-2016-0111. Epub 2016 Jun 08.

Opioids interact with sympathetic and renin-angiotensin systems in control of mean arterial pressure (MAP). Our earlier finding that biphalin, a synthetic enkephalin analogue, decreased MAP in anaesthetized spontaneously hypertensive rats (SHR) prompted us to further explore this action, to...

Cite
Bądzyńska B, Lipkowski AW, Olszyński KH, et al. Different blood pressure responses to opioids in 3 rat hypertension models: role of the baseline status of sympathetic and renin-angiotensin systems. Can J Physiol Pharmacol. 2016;94(11):1159-1169doi: 10.1139/cjpp-2016-0111.
Bądzyńska, B., Lipkowski, A. W., Olszyński, K. H., & Sadowski, J. (2016). Different blood pressure responses to opioids in 3 rat hypertension models: role of the baseline status of sympathetic and renin-angiotensin systems. Canadian journal of physiology and pharmacology, 94(11), 1159-1169. https://doi.org/10.1139/cjpp-2016-0111
Bądzyńska, Bożena, et al. "Different blood pressure responses to opioids in 3 rat hypertension models: role of the baseline status of sympathetic and renin-angiotensin systems." Canadian journal of physiology and pharmacology vol. 94,11 (2016): 1159-1169. doi: https://doi.org/10.1139/cjpp-2016-0111
Bądzyńska B, Lipkowski AW, Olszyński KH, Sadowski J. Different blood pressure responses to opioids in 3 rat hypertension models: role of the baseline status of sympathetic and renin-angiotensin systems. Can J Physiol Pharmacol. 2016 Nov;94(11):1159-1169. doi: 10.1139/cjpp-2016-0111. Epub 2016 Jun 08. PMID: 27494747.
Copy Download .nbib Format: NLM
Systemic study of selected histone deacetylase inhibitors in cardiac complications associated with cancer cachexia.

Canadian journal of physiology and pharmacology

Bora V, Patel D, Johar K, Goyal RK, Patel BM.
PMID: 34614370
Can J Physiol Pharmacol. 2021 Oct 06;1-12. doi: 10.1139/cjpp-2021-0012. Epub 2021 Oct 06.

Cancer cachexia is mainly characterized by wasting of skeletal muscles and fat and body weight loss, along with severe complications of major organs like liver, heart, brain and bone. There can be diminishing performance of these major organs as...

Cite
Bora V, Patel D, Johar K, et al. Systemic study of selected histone deacetylase inhibitors in cardiac complications associated with cancer cachexia. Can J Physiol Pharmacol. 2021;1-12doi: 10.1139/cjpp-2021-0012.
Bora, V., Patel, D., Johar, K., Goyal, R. K., & Patel, B. M. (2021). Systemic study of selected histone deacetylase inhibitors in cardiac complications associated with cancer cachexia. Canadian journal of physiology and pharmacology, 1-12. https://doi.org/10.1139/cjpp-2021-0012
Bora, Vivek, et al. "Systemic study of selected histone deacetylase inhibitors in cardiac complications associated with cancer cachexia." Canadian journal of physiology and pharmacology vol. (2021): 1-12. doi: https://doi.org/10.1139/cjpp-2021-0012
Bora V, Patel D, Johar K, Goyal RK, Patel BM. Systemic study of selected histone deacetylase inhibitors in cardiac complications associated with cancer cachexia. Can J Physiol Pharmacol. 2021 Oct 06;1-12. doi: 10.1139/cjpp-2021-0012. Epub 2021 Oct 06. PMID: 34614370.
Copy Download .nbib Format: NLM
The Fifth Tomoh Masaki Award (2019).

Canadian journal of physiology and pharmacology

D'Orléans-Juste P, Lapointe C.
PMID: 34459229
Can J Physiol Pharmacol. 2020 Sep;98(9):xiii-xiv. doi: 10.1139/cjpp-2020-0434. Epub 2020 Sep 17.

No abstract available.

Cite
D'Orléans-Juste P, Lapointe C. The Fifth Tomoh Masaki Award (2019). Can J Physiol Pharmacol. 2020;98(9):xiii-xivdoi: 10.1139/cjpp-2020-0434.
D'Orléans-Juste, P., & Lapointe, C. (2020). The Fifth Tomoh Masaki Award (2019). Canadian journal of physiology and pharmacology, 98(9), xiii-xiv. https://doi.org/10.1139/cjpp-2020-0434
D'Orléans-Juste, Pedro, and Lapointe, Catherine. "The Fifth Tomoh Masaki Award (2019)." Canadian journal of physiology and pharmacology vol. 98,9 (2020): xiii-xiv. doi: https://doi.org/10.1139/cjpp-2020-0434
D'Orléans-Juste P, Lapointe C. The Fifth Tomoh Masaki Award (2019). Can J Physiol Pharmacol. 2020 Sep;98(9):xiii-xiv. doi: 10.1139/cjpp-2020-0434. Epub 2020 Sep 17. PMID: 34459229.
Copy Download .nbib Format: NLM
Cyanidin-3-O-glucoside attenuates high glucose-induced podocyte dysfunction by inhibiting apoptosis and promoting autophagy via activation of SIRT1/AMPK pathway.

Canadian journal of physiology and pharmacology

Wang S, Huang Y, Luo G, Yang X, Huang W.
PMID: 33049148
Can J Physiol Pharmacol. 2021 Jun;99(6):589-598. doi: 10.1139/cjpp-2020-0341. Epub 2020 Oct 13.

Diabetic nephropathy (DN) is a common and complicated chronic kidney disease around the world. To elucidate and find effective therapies of DN is of vital importance. In this paper, we have discovered that cyanidin-3-O-glucoside (C3G), which is one of...

Cite
Wang S, Huang Y, Luo G, et al. Cyanidin-3-O-glucoside attenuates high glucose-induced podocyte dysfunction by inhibiting apoptosis and promoting autophagy via activation of SIRT1/AMPK pathway. Can J Physiol Pharmacol. 2020;99(6):589-598doi: 10.1139/cjpp-2020-0341.
Wang, S., Huang, Y., Luo, G., Yang, X., & Huang, W. (2021). Cyanidin-3-O-glucoside attenuates high glucose-induced podocyte dysfunction by inhibiting apoptosis and promoting autophagy via activation of SIRT1/AMPK pathway. Canadian journal of physiology and pharmacology, 99(6), 589-598. https://doi.org/10.1139/cjpp-2020-0341
Wang, Shu, et al. "Cyanidin-3-O-glucoside attenuates high glucose-induced podocyte dysfunction by inhibiting apoptosis and promoting autophagy via activation of SIRT1/AMPK pathway." Canadian journal of physiology and pharmacology vol. 99,6 (2021): 589-598. doi: https://doi.org/10.1139/cjpp-2020-0341
Wang S, Huang Y, Luo G, Yang X, Huang W. Cyanidin-3-O-glucoside attenuates high glucose-induced podocyte dysfunction by inhibiting apoptosis and promoting autophagy via activation of SIRT1/AMPK pathway. Can J Physiol Pharmacol. 2021 Jun;99(6):589-598. doi: 10.1139/cjpp-2020-0341. Epub 2020 Oct 13. PMID: 33049148.
Copy Download .nbib Format: NLM
Effect of ivabradine in heart failure: a meta-analysis of heart failure patients with reduced versus preserved ejection fraction.

Canadian journal of physiology and pharmacology

Tóth N, Soós A, Váradi A, Hegyi P, Tinusz B, Vágvölgyi A, Orosz A, Solymár M, Polyák A, Varró A, Farkas AS, Nagy N.
PMID: 34636643
Can J Physiol Pharmacol. 2021 Nov;99(11):1159-1174. doi: 10.1139/cjpp-2020-0700. Epub 2021 Oct 12.

In clinical trials of heart failure reduced ejection fraction (HFrEF), ivabradine seemed to be an effective heart rate lowering agent associated with lower risk of cardiovascular death. In contrast, ivabradine failed to improve cardiovascular outcomes in heart failure preserved...

Cite
Tóth N, Soós A, Váradi A, et al. Effect of ivabradine in heart failure: a meta-analysis of heart failure patients with reduced versus preserved ejection fraction. Can J Physiol Pharmacol. 2021;99(11):1159-1174doi: 10.1139/cjpp-2020-0700.
Tóth, N., Soós, A., Váradi, A., Hegyi, P., Tinusz, B., Vágvölgyi, A., Orosz, A., Solymár, M., Polyák, A., Varró, A., Farkas, A. S., & Nagy, N. (2021). Effect of ivabradine in heart failure: a meta-analysis of heart failure patients with reduced versus preserved ejection fraction. Canadian journal of physiology and pharmacology, 99(11), 1159-1174. https://doi.org/10.1139/cjpp-2020-0700
Tóth, Noémi, et al. "Effect of ivabradine in heart failure: a meta-analysis of heart failure patients with reduced versus preserved ejection fraction." Canadian journal of physiology and pharmacology vol. 99,11 (2021): 1159-1174. doi: https://doi.org/10.1139/cjpp-2020-0700
Tóth N, Soós A, Váradi A, Hegyi P, Tinusz B, Vágvölgyi A, Orosz A, Solymár M, Polyák A, Varró A, Farkas AS, Nagy N. Effect of ivabradine in heart failure: a meta-analysis of heart failure patients with reduced versus preserved ejection fraction. Can J Physiol Pharmacol. 2021 Nov;99(11):1159-1174. doi: 10.1139/cjpp-2020-0700. Epub 2021 Oct 12. PMID: 34636643.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 220 entries